5 edition of Hypothermia and cerebral ischemia found in the catalog.
Includes bibliographical references and index.
|Statement||edited by Carolina M. Maier and Gary K. Steinberg.|
|Contributions||Maier, Carolina M., Steinberg, Gary K.|
|LC Classifications||RC388.5 .H97 2004|
|The Physical Object|
|Pagination||x, 188 p. :|
|Number of Pages||188|
|ISBN 10||089603660X, 1592596533|
|LC Control Number||2003049949|
Sale Cerebral Ischemia and. Cerebral Ischemia and Calcium (English) Paperback Book Free Shipping! $ Cerebral ischemia that occurs within 6 hours of the operation (1 patient) is treated with prompt surgery and antegrade cerebral blood flow with or without the performance of a total arch replacement. The management of visceral ischemia (16 patients) has undergone a significant change in management over the course of this by: 7. The greatest protection was observed in the 30°C hypothermia group. Since a temperature of 30°C can be induced in humans by surface cooling without coagulopathy or ventricular fibrillation, hypothermia to 30°C may have potential clinical value for treatment of human brain by: ORIGINAL RESEARCH Catheter based selective hypothermia reduces stroke volume during focal cerebral ischemia in swine Thomas K Mattingly,1 Lynn M Denning,1 Karen L Siroen,1 Barb Lehrbass,2 Pablo Lopez-Ojeda,1,2 Larry Stitt,3 David M Pelz,1,2 Sumit Das,4 Lee-Cyn Ang,4 Donald H Lee,1,2 Stephen P Lownie1,2 For numbered afﬁliations seeCited by:
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In Hypothermia and Cerebral Ischemia: Mechanisms and Clinical Applications, leading investigators and pioneers from around the world take hypothermia from bench to bedside, comprehensively reviewing both the scientific and clinical studies that have led to its resurgence in the treatment of stroke.
Topics of special interest include Format: Hardcover. Read "Hypothermia and Cerebral Ischemia Mechanisms and Clinical Applications" by available from Rakuten Kobo.
A comprehensive review of the scientific and clinical studies that have led to the resurgence of interest in hypothermia Brand: Humana Press. In Hypothermia and Cerebral Ischemia: Mechanisms and Clinical Applications, leading investigators and pioneers from around the world take hypothermia from bench to bedside, comprehensively reviewing both the scientific and clinical studies that have led to its resurgence in the treatment of stroke.
In Hypothermia and Cerebral Ischemia: Mechanisms and Clinical Applications, leading investigators and pioneers from around the world take hypothermia from bench to bedside, comprehensively reviewing both the scientific and clinical studies that have led to its resurgence in the treatment of : Humana Press.
Clinical trials of hypothermia after cardiac arrest (global cerebral ischaemia) The experimental hypothermia studies in animal models of global cerebral Hypothermia and cerebral ischemia book paved the way for several clinical trials conducted from late to (Table 1).Two medium sized phase III trials using surface cooling showed a similar degree of improvement in outcome (55% vs 39%) and Cited by: Recently, Hong et al.
also showed that 48 h of mild hypothermia and 48 h of rewarming following acute ischemic stroke led to a lower incidence of hemorrhagic transformation, a better outcome measured by the modified Rankin Score, and a lesser degree of malignant cerebral edema.
This study had a much larger number of patients treated with 75 Cited by: Hypothermia and Cerebral Ischemia: Mechanisms and Clinical Applications Softcover PDF DOwnload by Carolina M. Maier (Editor), Gary K.
Steinberg (Editor) A comprehensive review of the scientific and clinical studies that have led to the resurgence of interest in hypothermia as a neuroprotective strategy in the treatment of stroke and traumatic brain injury.
Hypothermia and Cerebral Ischemia: Mechanisms and Clinical Applications. Maier and G. Steinberg (editors). Published by Humana Press, Totowa, New JerAuthor: N.M. Dearden. Therapeutic hypothermia has been widely to be one of the most reliable neuroprotective therapies for several cerebral disorders and injuries (van der Worp et al.,Yenari and Han, ), including stroke, traumatic injury, global ischemia after cardiac arrest, and hypoxic-ischemic by: Lee "Hypothermia and Cerebral Ischemia Mechanisms and Clinical Applications" por disponible en Rakuten Kobo.
A comprehensive review of the scientific and clinical studies that have led to the resurgence of interest in hypothermia Brand: Humana Press. During hypothermia, oxygen is redistributed in the brain.
3 The cerebral metabolic rate, cerebral ICP, and neurotoxic sequelae of ischemia all decrease. Cerebral blood flow decreases with alpha-stat pH management of ABG but is maintained with pH-stat management (see D).
Huang F, Zhou L. Effect of mild hypothermia on the changes of cerebral blood flow, brain blood barrier and neuronal injuries following reperfusion of focal cerebral ischemia in rats. Chin Med J (Engl). ISBN: X OCLC Number: Description: x, pages: illustrations ; 24 cm: Contents: Resurgence of hypothermia as a treatment for brain injury / Carolina M.
Maier and Gary K. Steinberg --The effects of hypothermia and hyperthermia in global cerebral ischemia / Myron D. Ginsberg and Ludmila Belayev --Mild. Hypothermia and Cerebral Ischemia: Mechanisms and Clinical Applications is intended to provide a comprehensive review of mild hypothermia, including the therapeutic potential of the methodology, limitations, and recent developments in both basic and clinical : Patricia H.
Petrozza. Get this from a library. Hypothermia and cerebral ischemia: mechanisms and clinical applications. [Carolina M Maier; Gary K Steinberg;] -- Hypothermia is one of the most effective neuroprotective therapies in experimental ischemia modes, and there is widespread interest in using it to treat stroke and traumatic brain injury (TBI).
Hypothermia and Cerebral Ischemia contains a plethora of up-to-date and important information on both basic and clinical research.
It is worthwhile to recommend as a reference book for the student, scientist and physician who is interested in or involved in the study of mild hypothermia for the treatment of stroke and traumatic brain injury. In this study we compared the efficacy of mild (35°C) and moderate (33°C) hypothermia alone and when combined with magnesium in a transient focal cerebral ischemia rat model.
Cerebral Ischemia and Post-Ischemic Treatment with Hypothermia. By Kym Campbell, Neville W. Knuckey and Bruno P. Meloni. Submitted: March 31st Reviewed: September 5th Published: March 2nd DOI: /Author: Kym Campbell, Neville W.
Knuckey, Bruno P. Meloni. Mechanisms affected by therapeutic hypothermia on the sub-acute stage of ischemic stroke. Ischemia activates both intrinsic and extrinsic pathway which will lead cell apoptosis.
Cerebral ischemia can occur hours after DHCA, because CBF and CVR are deranged for several hours after temperatures return to normal. Circulate cold blood minutes prior to rewarming (wash out metabolites) and consider selective cerebral perfusion if more than one hour of operative time is needed.
A new rectal cooling device for therapeutic hypothermia (TH) therapy is designed and is applied in TH treatment of SD rats with ischemic-hypoxic brain damage. Healthy adult SD rats (n = 45) were randomly assigned into four groups: the healthy control group (n = 5), the ischemia and hypoxia group (n = 10), the rectal TH cooling group (n = 18), and the ice blanket Cited by: 1.
Clinical studies of induced hypothermia were conducted on humans based on successful cerebral ischemia animal models. One of the first studies cooled 17 patients with stroke admitted within 12 hours from symptom onset (mean hours) for 6 hours.[ 43 ]Author: Edgar A.
Samaniego. Neuroprotection in transient focal cerebral ischemia by combination drug therapy and mild hypothermia: comparison with customary therapeutic regimen. Stroke, 6:  Zausinger S, Scholler K, Plesnila N and Schmid-Elsaesser R.
Combination drug therapy and mild hypothermia after transient focal cerebral ischemia in by: 5. Furthermore, in experimental models of transient cerebral ischemia, moderate-to-mild hypothermia can reduce infarct volume by more than 50%.
 However, in permanent MCAo models the. Hypoxia–ischemia before or around the time of birth occurs in approximately 2/ live births and is associated with a high risk of death or lifelong disability. Therapeutic hypothermia is now well established as standard treatment for infants with moderate to severe hypoxic–ischemic encephalopathy but is only partially effective.
There is compelling preclinical and clinical Cited by: Mild Hypothermia Mitigates Post-Ischemic Neuronal Death Following Focal Cerebral Ischemia in Rat Brain: Immunohistochemical Study of Fas, Caspase-3 and TUNEL. Phanithi PB, Yoshida Y, Santana A, et. Mild hypothermia in the treatment of severe cerebral ischemia using cooling blankets is safe and does not lead to severe side effects.
Mild hypothermia can help to control critically elevated ICP values in severe space-occupying stroke and may improve clinical outcome in these by: Hypothermia prevents the delayed decline in phosphocreatine and adenosine triphosphate, as well as the simultaneous increase in cerebral lactate concentration, seen 8–12 hours after hypoxia–ischemia in newborn piglets 29 However, it is not clear if preservation of energy metabolism in the delayed phase of injury is the primary mechanism Cited by: This book, with a largely North American authorship and contributions from Germany and Japan, comprises 10 chapters that address experimental and clinical studies of mild hypothermia (reductions of 2 to 5° below normal brain temperature) in global and focal cerebral ischaemia, and in traumatic brain : A J Strong.
By Carolina M. Maier, Gary K. Steinberg. A complete evaluate of the medical and scientific reports that experience resulted in the resurgence of curiosity in hypothermia as a neuroprotective approach within the remedy of stroke and stressful mind harm.
issues of unique significance contain intraoperative and extensive care administration of hypothermia-treated sufferers, a /5(4). Brain ischemia is a condition in which there is insufficient blood flow to the brain to meet metabolic demand. This leads to poor oxygen supply or cerebral hypoxia and thus to the death of brain tissue or cerebral infarction / ischemic stroke.
It is a sub-type of stroke along with subarachnoid hemorrhage and intracerebral hemorrhage. Ischemia leads to alterations in brain metabolism Other names: Cerebral ischemia, Cerebrovascular ischemia. An animal study comparing mild and moderate TH showed that 33°C was better tolerated than 30°C in a rat model of transient focal cerebral ischemia.
Another study showed that treatment with hypothermia of °C in the reperfusion period of focal cerebral ischemia is superior to all other applied by: Application of a novel rectal cooling device in hypothermia therapy after cerebral hypoxia-ischemia in rats Peng Liu1,2, Rui Yang1 and Zelan Zuo1* Abstract Background: A new rectal cooling device for therapeutic hypothermia (TH) therapy is designed and is applied in TH treatment of SD rats with ischemic-hypoxic brain by: 1.
Deep hypothermic circulatory arrest (DHCA) is a surgical technique that induces deep medical involves cooling the body to temperatures between 20 °C (68 °F) to 25 °C (77 °F), and stopping blood circulation and brain function for up to one hour.
It is used when blood circulation to the brain must be stopped because of delicate surgery within the brain, or. effects, the benefit of hypothermia in animal models is more consistent and robust than that of any other treatment strategy.
In a systematic review and meta-analysis of animal studies of focal cerebral ischemia, including data obtained from a total of 3, animals, hypothermia reduced the infarct size by 44% (95% confidence interval, 40% to Cited by: "Effects of Hypothermia on Local Blood Flow and Metabolism During Cerebral Ischemia and Hypoxia" published on Mar by Journal of Neurosurgery Publishing by: Blood-brain barrier (BBB) disruption is thought to facilitate the development of cerebral infarction after a stroke.
In a typical stroke model (such as the one used in this study), the early phase of BBB disruption reaches a peak 6 h post-ischemia and largely recovers after 8–24 h, whereas the late phase of BBB disruption begins 48–58 h by: 6.
Title:Combination of Therapeutic Hypothermia and Other Neuroprotective Strategies after An Ischemic Cerebral Insult VOLUME: 12 ISSUE: 5 Author(s):Joline Goossens and Said Hachimi-Idrissi Affiliation:Critical Care Department and Cerebral Resuscitation Research Group, Ghent University, De PintelaanGhent, Belgium.
Keywords:Acute ischemic stroke, clinical. Ongoing Clinical Trial Hypothermia in the Therapy of Ischemic Stroke Correlation of Hypothermia with Decrease of Glutathione Concentration and Tolerance to Cerebral Ischemia Hypothermia Prolongs the Viability of Ischemic Brain Tissue Due to Neuroprotection Linked to Redistribution of Oxygen in Brain: Positron Emission Tomography Study of the Price: $ Especially useful for bedside care in the ICU are the easy-reference care-management "cards" in the appendix of the book.
Brain Hypothermia Treatment provides a valuable resource and want to buy both, with priority on the volume. I used this book to help prepare for a visiting lecture I gave on Cerebral Ischemia: Cellular and Molecular 4/5(1). Accordingly, protracted hypothermia of degrees C may be beneficial following acute cerebral ischemia.
But the pathophysiological mechanism of this protection remains yet unclear. Although reduction of metabolism could explain protection by deep hypothermia, it does not explain the robust protection connected with mild hypothermia.book hypothermia ischemia research stroke TBI A comprehensive review of the scientific and clinical studies that have led to the resurgence of interest in hypothermia as a neuroprotective strategy in the treatment of stroke and traumatic brain injury (TBI).
The effect of hypothermia on neuronal injury following permanent middle cerebral artery (MCA) occlusion in the rat was examined.
Moderate hypothermia (body temperature 24°C) was induced before MCA occlusion (0-minute delay group) in six rats, at 30 minutes in eight rats, and at 1 (seven rats), 2 (seven rats), and 3 (nine rats) hours after by: